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Which side will the tongue deviate in a lesion of the hypoglossal nerve?

Which side will the tongue deviate in a lesion of the hypoglossal nerve?

weak side
This is solely responsible for the musculature of the tongue. Isolated damage to one hypoglossal nerve causes unilateral wasting of the tongue and the tongue deviates towards the weak side on protrusion due to the unopposed action of the muscle on the normal side.

What happens when the hypoglossal nerve is damaged?

The hypoglossal nerve can be damaged at the hypoglossal nucleus (nuclear), above the hypoglossal nucleus (supranuclear), or interrupted at the motor axons (infranuclear). Such damage causes paralysis, fasciculations (as noted by a scalloped appearance of the tongue), and eventual atrophy of the tongue muscles.

What nerve causes tongue deviation?

Damage to the hypoglossal nerve causes paralysis of the tongue. Usually, one side of the tongue is affected, and when the person sticks out his or her tongue, it deviates or points toward the side that is damaged.

What nerve controls tongue movement?

The hypoglossal nerve enables tongue movement. It controls the hyoglossus, intrinsic, genioglossus and styloglossus muscles.

Can the hypoglossal nerve repair itself?

From cases reported in the literature, complete recovery of hypoglossal nerve function is expected within the first six months.

Does Bell’s palsy cause tongue deviation?

1. Illustrates neurologic signs in patients diagnosed as having Bell’s palsy. 1, intact fore- head; 2, miosis ; 3, loss of corneal sensation ; 4, loss of tearing ; 5, loss of sensation ; 6, deviation of tongue; and 7, loss of taste papillac.

What causes nerve damage in tongue?

Damage to the lingual nerve occurs most commonly when removing a wisdom tooth, also known as the third molar, in the lower jaw. This can lead to a feeling of numbness, a prickling sensation, and sometimes a change in how food or drink tastes.

How do you fix nerve damage in the tongue?

Supportive psychotherapy with steroids, antidepressants, and anticonvulsants may be used to treat lingual nerve injury. Most cases of lingual injuries recover within 3 months without special treatment, but some patients have reported permanent lingual nerve injury [9].

Is tongue paralysis curable?

How is paralysis treated? Currently, there is no cure for paralysis itself. In certain cases, some or all muscle control and feeling returns on its own or after treatment of the cause for the paralysis. For example, spontaneous recovery often occurs in cases of Bell’s palsy, a temporary paralysis of the face.

How do you test for hypoglossal nerve damage?

The hypoglossal nerve is tested by examining the tongue and its movements. At rest, if the nerve is injured a tongue may appear to have the appearance of a “bag of worms” (fasciculations) or wasting (atrophy). The nerve is then tested by sticking the tongue out.

What causes tongue deviation in a unilateral stroke?

Tongue deviation in unilateral stroke most likely results from asymmetrical supranuclear control of the 12th cranial nerve in many individuals. The finding that it occurs relatively commonly in large (non-lacunar) infarcts and its association with dysphagia may have clinical utility.

Is the tongue a symptom of a stroke?

This is called tongue deviation [2-5]. Hence, the symptom of tongue deviation is observed in a stroke or TIA [5-8]. For thousands of years, the deviation of the tongue has also been recognized as a symptom of what is called a “wind stroke” in traditional Chinese medicine [9-11].

What kind of nerve palsy causes tongue deviation?

All tongue deviations were associated with supranuclear 7th nerve palsy. Dysphagia and dysarthria occurred in 43 and 90% of patients with tongue deviation.

What causes tongue protrusion in the midline?

Lesions of the hypoglossal nerve can be divided into supranuclear, nuclear, and infranuclear according to the lesion site. To protrude the tongue in the midline, a balanced action of both genioglossus muscles is required. A supranuclear palsy will cause motor dysfunction contralateral to the side of the lesion without atrophy or fasciculations.